By Ralph Boulton, Sanjeev Gupta, Claire Cousins, Humphrey Hodgson
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Extra resources for A Colour Handbook of Gastroenterology
Pylori infection H. pylori colonizes the epithelium of the gastric antrum in chronic infection (65). The organism lies adjacent to the gastric epithelial cells, and in the gastric pits beneath the mucous layer. Active, chronic gastritis develops predominantly in the antrum. Degenerative changes occur in the epithelium, and chronic inflammatory cells consisting of polymorphs, plasma cells, and 65 H. pylori seen at the surface of the gastric epithelium. lymphocytes infiltrate the mucosa. Lymphoid aggregation and follicle formation also occur in the basal mucosa.
Staphylococcus aureus toxins). 62 Haemorrhagic gastritis with mild patchy erythema: (A), antegrade view; (B) in retroversion. 63 Clinical history and examination Patients complain of abdominal pain, anorexia, nausea, retching, and gastrointestinal bleeding. In the setting of the intensive therapy unit (ITU) patient, these lesions are a frequent cause of GI bleeding – shallow erosions present with bleeding from the capilliary plexus. Laboratory and special examinations The mucosa is erythematous and congested at endoscopy.
Subsequent tumour overgrowth of the stent can cause obstruction, which can usually be controlled by endoscopic means. 24 Palliation of oesophageal cancer with a self-expanding metal stent. This sequence shows placement of a guidewire across the cancer and then deployment of the stent. 24E Achalasia and other primary motility disorders 31 Achalasia and other primary motility disorders The motility disorders of the oesophagus include inadequate lower oesophageal sphincter relaxation (typified by achalasia), uncoordinated contraction and hypercontraction (such as diffuse oesophageal spasm), and hypocontraction with ineffective oesophageal motility.
A Colour Handbook of Gastroenterology by Ralph Boulton, Sanjeev Gupta, Claire Cousins, Humphrey Hodgson